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Health & Fit Drug Could Reduce Heart Disease Risk by Targeting Gut

22:15  06 august  2018
22:15  06 august  2018 Source:   newsweek.com

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of drug which could reduce the risk of cardiovascular disease by harnessing our gut bacteria. A new class of drug could decrease the risk of cardiovascular disease by targeting a specific FDA recalls heart drug over cancer concerns. When the gut digests nutrients present in animal products

By preventing gut bacteria from producing a particular chemical, it might be possible to reduce cardiovascular risk . A new drug that blocks the production of one chemical by gut bacteria significantly reduces cardiovascular Blocking a gut bacteria enzyme could improve heart health.

Scientists have developed a new class of drug which could cut the risk of cardiovascular disease by harnessing our gut bacteria.

The compound developed by scientists at Cleveland Clinic can stop gut microbes from making a molecule which has been linked to heart disease without harming them. 

The research is the latest to investigate the role of the microbiome on our overall physical health. This is the genetic makeup of the bacteria which populates the inside and outside of our bodies and outnumber our own cells by 10 to 1. 

Dr. Stanley Hazen of the Cleveland Clinic department of cardiovascular medicine and author of the study published in the journal Nature Medicine ​said in a statement: "To our knowledge, this is the most potent therapy to date for 'drugging' the microbiome to alter a disease process."

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Heart disease is often linked with atherosclerosis. Until now, research efforts to lower TMAO levels in hopes of decreasing heart disease risk This new finding suggests that drugging these microbes — together known as the gut microbiome — could help treat a variety of disorders , the researchers said.

"The approach developed could potentially be used to target other gut microbial pathways. We look forward to advancing this novel therapeutic strategy Heart disease is the leading cause of death for both men and women in the United States. Every year, about 610,000 people die of heart disease in

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To develop the drug, researchers carried out experiments in mice. They found the compound they created made platelets (cell fragments in the blood which help with clotting) less overly responsive and also prevented excessive blood clots. Both of these are risk factors for cardiovascular disease.

The drug achieved this by reducing levels of trimethylamine N-oxide (TMAO), a byproduct of digestion, in the blood. 

Previous studies headed by the same team indicated high levels of TMAO in the blood could be a way for predicting if a person will suffer a heart attack and stroke.

When the gut digests nutrients present in animal products such red meat, egg yolks, and high-fat dairy products, it release TMAO. This in turn can affect how platelets behave, and raise the risk of thrombosis which can cause heart attacks and stroke.

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Researchers have designed a potential new class of drugs that may help reduce the risks of cardiovascular disease by targeting a specific microbial pathway in the gut . Unlike antibiotics, which non-specifically kill gut bacteria and can lead to adverse side effects and resistance, the new class of

“The approach developed could potentially be used to target other gut microbial pathways. We look forward to advancing this novel therapeutic strategy Heart disease is the leading cause of death for both men and women in the United States. Every year, about 610,000 people die of heart disease in

One dose of the drug cut the levels of TMAO in the blood for up to three days. It also returned platelets to their normal levels of responsiveness and made clots less likely to form after damage to the arteries. The drug lingered in the gut, but didn’t harm important bacteria - meaning it won’t put patients at risk of antibiotic resistance.

Hazen said: “Gut bacteria are altered [by the drug] but not killed by this drug, and there were no observable toxic side effects. The approach developed could potentially be used to target other gut microbial pathways."

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